英语翻译Oxidative stress mediated by Nox1- and Nox4-based NADPH oxidase (Nox) plays a key role in vascular diseases.The molecular mechanisms involved in the regulation of Nox are not entirely elucidated.Because JAK/STAT regulates many genes linke

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英语翻译Oxidative stress mediated by Nox1- and Nox4-based NADPH oxidase (Nox) plays a key role in vascular diseases.The molecular mechanisms involved in the regulation of Nox are not entirely elucidated.Because JAK/STAT regulates many genes linke
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英语翻译Oxidative stress mediated by Nox1- and Nox4-based NADPH oxidase (Nox) plays a key role in vascular diseases.The molecular mechanisms involved in the regulation of Nox are not entirely elucidated.Because JAK/STAT regulates many genes linke
英语翻译
Oxidative stress mediated by Nox1- and Nox4-based NADPH oxidase (Nox) plays a key role in vascular diseases.The molecular mechanisms involved in the regulation of Nox are not entirely elucidated.Because JAK/STAT regulates many genes linked to inflammation,cell proliferation,and differentiation,we questioned whether this pathway
is involved in the regulation of Nox1 and Nox4 in human aortic smooth muscle cells

英语翻译Oxidative stress mediated by Nox1- and Nox4-based NADPH oxidase (Nox) plays a key role in vascular diseases.The molecular mechanisms involved in the regulation of Nox are not entirely elucidated.Because JAK/STAT regulates many genes linke
氧化应激,Nox4-based介导的Nox1 NADPH氧化酶(Nox)中扮演了重要的角色血管疾病.分子生理机制Nox的规定尚未完全阐明.因为JAK /属性调节许多基因与炎症、细胞增殖和分化,这条路,我们质疑
参与的规定和Nox4 Nox1人类主动脉平滑肌细胞

氧化应激介导的NOX 1-nox4-basedNAD PH氧化酶(氮氧化物)中起着关键作用血管疾病。分子机制参与调节氮并不完全阐明。因为激酶/转录调节许多基因与炎症,细胞增殖,分化,我们质疑这一途径是参与调节NOX 1和及其在人主动脉平滑肌细胞